Alias: | ;N-Methyl-N'-nitro-N''-[(tetrahydro-3-furanyl)Methyl]guanidine | Purity: | 99% |
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CAS NO.: | 165252-70-0 | MW: | 202.21 |
Type: | Insecticides. Organochlorine Insecticides | Appearance: | White To Pale Brown Powder |
Highlight: | Raw Materials Pesticide Dinotefuran,Pesticide Dinotefuran 165252-70-0 |
Pesticide Intermediate Dinotefuran Powder With CAS NO. 165252-70-0
Dinotefuran acts through contact and ingestion and results in the cessation of feeding within several hours of contact and death shortly after. Dinotefuran does not inhibit cholinesterase or interfere with sodium channels. Therefore, its mode of action is different from those of organophosphate, carbamate, and pyrethroid compounds. It appears that Dinotefuran acts as an agonist of insect nicotinic acetylcholine receptors, but it is postulated that Dinotefuran affects the nicotinic acetylcholine binding in a mode that differs from other neonicotinoid insecticides. It is reported that Dinotefuran was highly active on a certain silverleaf whitefly strain which developed resistance against imidacloprid.
ITEM | CONTENT |
Alias | N''-Methyl-N-nitro-N'-[(tetrahyChemicalbookdro-3-furanyl)Methyl]guanidine |
Density | 1.42±0.1 g/cm3(Predicted) |
CAS No. | 165252-70-0 |
color | White to Pale Brown |
Purity | 99% |
Sensitive | Light Sensitive |
EINECS No. | 1806241-263-5 |
Storage | 0-6ºC |
MF | C7H14N4O3 |
Solubility in water | 39.83 g/L |
Type | Pesticide raw materials |
Boiling Point | 334.5±34.0 °C(Predicted) |
Sensitive | Light Sensitive |
Dinotefuran is a neonicotinoid which is a chemical designed to mimic the effects of nicotine, found commonly in tobacco. Dinotefuran is also systemic, meaning that it is absorbed into the plants that it is applied on and moves through the entire plant body down to the root. Dinotefuran's mode of action is achieved by disrupting stimuli transmission within the nervous system of the targeted insect when the insect either ingests or absorbs the active into its body.
Dinotefuran blocks certain neuronal pathways that are much more common in insects than mammals. That is why the chemical is much more toxic to insects than humans or animals. As a result of this blockage, the insect begins to overproduce acetylcholine, an important neurotransmitter, resulting in the insect's paralysis, and eventually their death.